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The AAV-PPAR treatment markedly reduced hepatic fibrosis, as evidenced by decreased -SMA immunoreactivity, COL1A1 expression and Sirius red staining, with these antifibrotic effects further confirmed by consistent downregulation of -SMA and Col1a1 at both the protein and transcript levels (Fig

Areas of Research Wound repair and tissue remodeling research Angiogenesis and vascular biology research Tendon, ligament, and connective tissue studies Gastrointestinal mucosal and barrier research Enteric nervous system and gut-brain axis research Research Use Only: This product is sold exclusively for laboratory and scientific research

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