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Alcohol has a very similar mechanism of tolerance and withdrawal to benzodiazepines, involving the GABAA, NMDA, and AMPA receptors [237], and thereby, the extent of this counteraction should be indicative of the observed effects [63,64]

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Second, the EEG endpoint that defined the compounds activity is a population-level pattern in the recipient cortex, not a molecular readout meaning the discovery established a phenomenology, not a pathway

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