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Overall, the benefits of aerobic exercise are limited by the functional reserve beyond the mutation load in MM patients, and the magnitude of improvement is largely determined by the underlying genotype ( MM patients experience exercise intolerance and delayed post-exercise recovery as a result of defective oxidative phosphorylation, which limits ATP production ( MM patients typically exhibit elevated oxidative stress due to ETC defects, resulting in excessive ROS production that damages mtDNA

P., Leu, N

Fish Diseases, 35, 799-808

A significant CAR effect (p=0.038) resulted from increased numbers of phagocytosing PMN 0.5 until 72 h pp in CAR supplemented cows compared to non-supplemented cows