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The NLRP3 inflammasomes are central to the initiation of inflammatory responses by activating caspase-1 and promoting the release of pro-inflammatory cytokines, particularly interleukin (IL)-1 and IL-18

(2009) Antimony-trioxide- and arsenic-trioxide-induced apoptosis in myelogenic and lymphatic cell lines, recruitment of caspases, and loss of mitochondrial membrane potential are enhanced by modulators of the cellular glutathione redox system

Additionally, BVA therapy had a strong antihyperalgesic impact at acupoint ST36 (Yoon et al., 2012)

Centrally administrated urocortins are also able to suppress ghrelin secretion, potentially preventing ghrelin-induced stimulation of appetite (Yakabi et al., 2011)