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Elucidating the detailed molecular mechanisms by which AMPARs traffic to and away from synapses is an active topic of current investigations (see West and Greenberg 2012)

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Deficiency in the level of activity of glucose-6-phosphate dehydrogenase (G6PD) is the basis of favism, primaquine (an anti-malarial drug) sensitivity and several other drug-sensitive hemolytic anemias, anemia and jaundice in the newborn, and chronic nonspherocytic hemolytic anemia

All mice were euthanized on day 15