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For the first time stable isotope labelling during cell cultivation will be applied in the context of sulfur metabolomics
(2014) revealed a novel repair pathway for cPu lesions that results in trinucleotide repeat (TNR) deletion via a unique DNA polymerase (Pol) lesion bypass during DNA replication and BER
GSNO may be animportant physiological NO donor, for example, in the regulation of NPR1, where nitrosylation of proteine cysteine residues acts antagonistically to reduction by TRX (Tada et al., 2008)
