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Although other report denied the presence of any GST activity in the nucleus [18] various reports [19], [20] including a recent work by Stella et al
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Mechanistically, BPC-157 appears to modulate angiogenesis, upregulate growth factors such as VEGF and FGF, and influence nitric oxide pathways, contributing to enhanced vascularization, tissue repair, and anti-inflammatory effects
A role for mitochondria in ALS may explain why SOD1 mutant proteins selectively damage the motor neurons
