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This is possible due to the extraordinary ability of the gonococcus to modulate the interaction with different types of cervical epithelial cells in the female tract, using distinct mechanisms, including the modulation and control of distinct epithelial cell-cell adhesion complexes through the manipulation of host cell signaling and the ability to survive intracellularly within a gonococcal-containing vacuole (Edwards and Butler, 2011

Indeed some reports suggest that the GSTM1 genetic polymorphism may affect AFB 1 detoxification in human liver

Aldaoseri HA, Zubairi MB