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Studies suggest that various molecular pathways are involved in IC/BPS, including the JUN N-terminal kinase (JNK) pathway, transient receptor potential (TRP) channels, activated mast cells, mucosal signalling, circadian rhythm regulation, inflammation and neural dysregulation

Mitochondrial dysfunction in GDM is closely linked to increased production of reactive oxygen species (ROS), particularly in placental macrophages, which contributes to oxidative stress and cellular damage [100, 103]

Recently, a new isoprenylated sanggenon-type flavanone, nigrasin K, along with some other analogs including sanggenon M, C and O, chalcomoracin, sorocein H and kuwanon J isolated from the twigs of Morus nigra have been identified as potent tyrosinase inhibitors by Hu et al

According to studies of D2-like receptors, the modification of arrestin ubiquitination status is a key cellular event underlying receptor desensitization