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Selegiline reduces N-methyl-D-aspartic acid induced perturbation of neurotransmission but it leaves NMDA receptor dependent long-term potentiation intact in the hippocampus

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However, intracardiac treatment of Bnip3 / mice with B-017 five minutes before reperfusion provided no additional protection

Protease-activated receptors 1 and 4 mediate thrombin signaling in endothelial cells