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Death is usually due to cerebral edema, which causes an increase in intracranial pressure, and a reduced level of vigilance

Given the potential therapeutic efficacy of NAC against mTBI, we postulate that a short-term treatment with NAC might be helpful to modulate functional reorganization and to facilitate neuroplasticity through recovery profile in patients with post-concussion symptoms promising the enhancement in neuronal compensatory mechanism and neurocognitive performance

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(3) Iron homeostasis modulation: iron chelation with deferoxamine (DFO) and inhibition of NCOA4dependent ferritinophagy are illustrated as means to lower the labile iron pool and reduce ironcatalyzed lipid peroxidation