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The dual action creates a net negative fat balance: more fat is broken down, less new fat is formed (Ng et al., 2000)

Alongside providing adequate treatment, the patient should be followed up for a few weeks to examine for the resolution and screen for neurological manifestations, as there can be a late manifestation in some patients

Finding out how CnoX recognizes its substrates and what is the role, if any, played by the TPR domain in controlling substrate selectivity are other outstanding questions

Antipurinergic interventions based on suramin administration appear to act by inhibiting the excessive signaling of ATP and related nucleotides released from stressed cells