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The treatment increased QoL in patients and decreased systemic inflammation by inhibiting cytokines, such as TNF-, TGF-, IL-6, IL-8, hs-CRP, calcitonin gene-related peptide (CGRP), MCP-1, and substance P

After GSH is utilized, the oxidizing environment facilitates the reoxidation of protein thiols by interacting with protein disulfide isomerase (PDI) and endoplasmic reticulum oxidoreduction (ERO-1) (Higa and Chevet, 2012)

Each stack addresses a specific goal

3B), we found that the top keywords in the order of frequency and centrality were cell death (frequency: 129, centrality: 0.02), oxidative stress (frequency: 115, centrality: 0.11) and acute kidney injury (frequency: 81, centrality: 0.17)