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3367-3398, in The Metabolic and Molecular Bases of Inherited Disease , by Scriver C.R

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Overexpression of thioredoxin renders embryos more resistant to oxidative stress (Kobayashi-Miura et al., 2002), whereas thioredoxin null mutants exhibit dramatically reduced proliferation of the inner cell mass resulting in lethality during the early postimplantation period (reviewed in Ufer et al., 2010)