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Identification of inflammatory gene modules based on variations of human endothelial cell responses to oxidized lipids

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Liver disease itself can disrupt systemic and cellular magnesium homeostasis, while the resulting magnesium deficiency may subsequently act as a disease-modifying amplifier, exacerbating hepatic injury through mechanisms such as chronic inflammation, mitochondrial dysfunction, and oxidative stress ( Despite this compelling biological rationale, significant translational gaps persist

For example, dietary supplementation of CoQ10 positively influences FM symptoms