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Similarly, Baker and team affirmed that preexisting Toxoplasma gondii infection augments brain damage during TBI pathogenesis by modulating glutamatergic, neurotoxic, and oxidative stress markers, along with significant elevation in the expression of neuroinflammatory mediators [324]

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T., Emergence of the Th17 Pathway and Its Role in Host Defense, Advances in Immunology 99 (2008): 115163

Secondary mitochondrial diseases are more loosely defined and even harder to diagnose as they can arise from environmental triggers and mutations that do not directly encode mitochondrial function