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The washed immunoprecipitates were incubated in 50 L of 1 electrophoresis loading buffer and heated to 100 C for 5 min

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Abstract Background: The interplay between bacterial virulence factors and the host innate immune response in pneumococcal meningitis (PM) can result in uncontrolled neuroinflammation, which is known to induce apoptotic death of progenitor cells and post-mitotic neurons in the hippocampal dentate gyrus, resulting in cognitive impairment

Induction of MnSOD gene expression in a hepatic model of TNF- toxicity does not result in increased protein