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Therefore, in-depth research into the relationships among NRF2, PTMs, and epigenetic modifications, as well as their roles in regulating NRF2 function, is crucial for elucidating the molecular mechanisms governing cellular redox homeostasis

Nonetheless, extant clinical trials and vast research in animal models indicate a positive safety profile [17]

In addition, the treatments protected the BBB integrity and promoted neurological recovery in stroke model studies