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Moreover, G289A and T337I mutations are located proximal to the ATP-binding pocket of the C-terminal domain, G289A would cause steric conflict with Ser293, and T337I would cause a hydrophobic patch on the protein surface and reduce protein solubility
The osteoarthritic phenotypes have been found in multiple animal models used to study iron overload (Carroll, 2006
It is under active review by the FDA's PCAC advisory panel, with a formal evaluation expected in July 2026
M., Zynda, A
