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The pharmacokinetic analysis revealed that S-Acetyl-L-glutathione is rapidly deacetylated to GSH post-administration before it can reach the bloodstream, resulting in the non-quantifiable levels of plasma SAG

Blood-Based Biomarker GSH, GSSG, and GSH/GSSG Genetic variations in glutathione-related pathways have been observed in ASD (141144) and have been correlated to ASD behaviors in some studies (145, 146)

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Other mechanisms may also be important in bortezomib induced peripheral neuropathy, including nuclear accumulations of ubiquinated proteins and altered protein transcription in sensory ganglion neurons 67, 68