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It is well known that it is necessary to examine different parameters to evaluate the antioxidant capacity of a compound such as the interaction with other antioxidant molecules, the specificity in the elimination of free radicals, the metal-chelating ability, the outcomes on gene expression, the capability to repair oxidative injury, and the bioavailability [10]

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These pathways are implicated in amino acid turnover, redox balance, neurotransmitter regulation, and mitochondrial function, suggesting coordinated metabolic perturbations in ASD