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Schaan BD
Lipid-nanoparticle-based delivery of CRISPR/Cas9 genome-editing components
However, weeks after the onset of ischemia, the chronic phase leads to additional tissue damage and may be the result of delayed neurodegeneration triggered by oxidative stress and immune activation
In granulosa cells and murine embryonic stem cells, T-2 toxin treatment induces ROS accumulation, resulting in reduction of mitochondrial transmembrane potential
