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Description
Structural basis for cofactor and substrate selection by cyanobacterium succinic semialdehyde dehydrogenase
Tyrosinase inhibition by hemiindigoid-based compounds with a copper-interacting hydroxamic acid head
Clinical trials demonstrated remarkable results
This specificity was not only observed at the physiological level but also at the level of gene expression where we identified a unique hypothalamic gene expression signature associated with AP-induced hyperphagia that was fully blocked by co-treatment with minocycline
