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It is therefore possible to predict that CXCL9, CXCL10, and CXCL11 may interact with CXCR3-A and activate its downstream signaling cascades, which would then decrease the expression of CXCR3-B at the posttranscriptional or posttranslational level

these glutathione molecules can then function again as antioxidants, scavenging reactive oxygen species from the cell