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Specific accessory structures also contribute to the pathogenesis of NFGNB, such as secretion systems, which are utilized by many bacterial pathogens to transport various protein factors (e.g., adhesins, exotoxins, and exoenzymes) onto the bacterial surface, into the surrounding environment, or directly into host cells, disrupting the normal cellular functions and promoting infection [2]

Together, these findings have made BPC-157 a recurring experimental tool for studying nitric oxide signaling, endothelial activation, and vascular remodeling processes associated with tissue repair