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This impairment manifests as reduced mitochondrial biogenesis, dysregulated mitochondrial dynamics (imbalances in fusion and fission), and inefficient mitophagy, collectively accelerating EC dysfunction, senescence, and atherosclerotic progression [161,162,163,164]
Redner A, Andreeff M , Miller DR, Steinherz P, Melamed M
Elution Elute the target proteins from the immobilized ligands by altering the buffer conditions
