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GHK-Cu is believed to be involved in multiple mechanisms of regeneration and protection in different tissues around the body

Jacovina and colleagues reported a unique in vivo mechanism suggesting that an annexin A2-deficient mouse rendered hyperhomocysteinemic by dietary means demonstrated impaired fibrinolysis, perivascular fibrin persistence, and attenuated angiogenesis (angiostasis) (4)

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After intratracheal inhalation, the nanoparticles predominantly distribute in lung tumors and target DCs and macrophages, robustly activating antitumor immunity and suppressing lung metastases