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Mechanisms of arterial stiffening: from mechanotransduction to epigenetics
Nicotinamide adenine dinucleotide phosphate oxidase (NOX) isoforms are major sources for endogenous ROS, multifarious NOX isoforms are localized to various cellular membranes and involved in many physiological or pathological events (31, 32)
This paper reviews the research progress of ferroptosis signaling pathways and ferroptosis inducers
This shift reduces its exposure to the ubiquitin-proteasome system (UPS), as the cytosolic form is more susceptible to ubiquitination and subsequent degradation ( Figure 1B )
