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recently reported that consumption of a high-fat diet did not increase cPT1 mRNA level [38]
Heart-specific SCOT knockout mice (in which terminal oxidation of -hydroxybutyrate is prevented) exhibit increased fatty acid oxidation (Schugar et al., 2014), revealing the interrelated characteristics of myocardial ketone oxidation and fatty acid oxidation
An acute glycolytic-excitotoxic crisis and nicotinamide adenine dinucleotide (NAD + ) depletion trigger neuroimmune dysfunction
J Appl Toxicol
