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If our data based on an intense and short exposure to HNE can be extended to an in vivo situation where low concentrations of HNE are produced on a long time scale, the present results suggest that although the amount of DNA adducts is low upon treatment by exogenous HNE, their formation could be favored upon oxidative stress
Ferroptosis, an iron-dependent form of lipid peroxidation, contributes to DKD pathogenesis in diabetic mice
Protecting the skin from environmental stressors
