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Despite advances in targeting the KRAS protein directly or indirectly [2, 3] and the advent of immunotherapy [4], effective therapies for KRAS -mutant lung cancer remain elusive [5, 6], arguing for a better understanding of KRAS -driven tumorigenesis and the development of novel therapeutic strategies to combat the disease

When the bodys compensatory mechanisms are overwhelmed, particularly in states of suboptimal health, compromised mitochondrial functionality is characterised by increased generation of reactive oxygen species (ROS) frequently associated with a chronification of the low-grade inflammation and development of follow-up pathologies

This approach takes advantage of epitalon's effects on pineal gland function and sleep-related hormone regulation

Le DSIP cible une troisieme dimension : la qualite du sommeil profond et l'adaptation au stress chronique