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We thus hypothesize that less MG is produced in tumors by concomitant lower oxidation of DHAP/G3P to MG, due to a more efficient lower glycolysis with highly active serine de novo/glutathione synthesis and active TCA, reducing the pool of available DHAP/G3P for MG formation

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Intrinsically, neurons are at risk for oxidative damage as their redox potential remains low due to a poor de novo synthesis of GSH caused by a weak expression of Nrf2 in neurons compared to astrocytes (Bolaos 2016)

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