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Moreover, sFASL has been shown to interact with immune cells such as T lymphocytes and macrophages, enhancing the production of pro-inflammatory cytokines and contributing to the chronic inflammatory milieu characteristic of MASLD [231, 232]
Collectively, random drift, developmental hierarchy, and microenvironmental cues shape a multidimensional dynamic pool of epigenetic heterogeneity, suggesting that subpopulations with potentially drug-resistant epigenetic traits may already exist at the onset of treatment
F., Cheung, E
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