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In milder forms, this fatigue may remain unnoticed for years, while in more severe cases it becomes evident early in life [3][8]
This pathway can control the expression of PEBP1, ERK1/2, GPX4, FTH1, ACSL4, and COX2, thereby enhancing the function of hippocampal astrocytes and microglial cells in CUMS model mice (Jiao et al., 2021)
and secondly, improving the quality of life, especially with respect to alleviating symptom severity and slowing the progression of physical function decline attributed to the disease [188]
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