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Description
Recently it has been shown that HO-1 not simply slow down, but reverse plaque progression from a vulnerable plaque to a more stable phenotype (Cheng et al., Autopsy examinations of human atherosclerotic lesions revealed that HO-1 expression correlates with plaque instability and the level of pro-inflammatory markers
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Absence of retbindin blocks glycolytic flux, disrupts metabolic homeostasis, and leads to photoreceptor degeneration
Sharif, I
