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Targeting the interaction between STING and Src using the STING N18 peptide inhibits Syk-mediated signal transduction and strengthens the hosts antifungal response, suggesting promising applications for developing antifungal therapeutics ( Apart from the TBK1 and cGAS-STING pathways, other host immune molecules also modulate anti- C

Consequently, intracellular Fe 2+ could not be oxidized to Fe 3+ and excreted from macrophages via FPN, which may be the potential mechanism of iron retention in plaques