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Among these, the ER has emerged as a central hub for initiating lipid peroxidation, supported by imaging and lipidomics data showing that oxidation events in the ER precede those at the plasma membrane 83

tuberculosis infections by increasing levels of GSH, improving redox homeostasis, and dampening the effects of TGF-

[ 13 C 6 , 15 N 2 ]-L-Cystine (CNLM-4244-H-PK) and [ 12 C 2 , 15 N]-GSH (CNLM-6245-50) were purchased from Cambridge Isotope Laboratory

Panda A, Qian F, Mohanty S, van Duin D, Newman FK, Zhang L, et al
