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Description
The animal study by McCormack et al

[3] CRP and ESR Modulation Chronic inflammation is the enemy of repair

promote myocardial hypertrophy and fibrosis through the regulating the oxidation process of pyruvate and fatty acids, affecting cardiac energy metabolism and mitochondrial function, and accelerating the development of HF [113], these results suggest that TMAO may be a potential target for the treatment of HF

Disposal, 15: 356-362
