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M.MurdochG
& Lerat, H
The evidence that OTA caused increased oxidative stress led to the expectation that gene expression profiles after exposure to OTA in animals would show the induction of many genes responsive to exposure to oxidative stress
The LRRK2 induced DA toxicity and degeneration could be abrogated by WT PINK1 (but not PINK1 mutations) and early treatment with -MT was able to reverse the pathologies in human neurons and TG Drosophila models with pathogenic LRRK2 mutations
