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The dose-dependent activation of the FAK-paxillin pathway increases tendon fibroblast migration and survival under oxidative stress
With high antigen-presenting capacity, they secrete reactive oxygen species (ROS) and proinflammatory cytokines, including IL-1, IL-6, IL-12, IL-18, IL-23, and TNF-, which modulate Th1-mediated antigen-specific inflammatory responses (85, 86)
A.et al (2018)
doi: 10.1016/S1474-4422(09)70068-7
