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The present results indicated that the antihypertensive effect of 1 AR-selective antagonists may be explained by their ability to hamper norepinephrine release through presynaptic modulation, in agreement with previous analyses showing that the hypotensive effect of AR antagonists, including atenolol, did not depend on cardio depression or suppression of renin secretion (31)
G.MurphyJ
In this approach, unreacted GSH reduces Cu(II)-neocuproine to form a highly colored Cu(I)-neocuproine complex, with absorbance changes that inversely correlate with GPx activity [19]
doi: 10.1016/j.phrs.2007.01.009
