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Epileptogenic injuries or seizures initiate the synthesis and secretion of proinflammatory molecules such as TNF-, IL-1, and High Mobility Group Box 1 (HMGB1) in glial cells which result in decreasing seizure threshold, which further contributes to seizure precipitation, and recurrence because of rapid changes in glutamate and -aminobutyric acid (GABA) receptor phosphorylation

[DOI] [PMC free article] [PubMed] [Google Scholar] 339.Wolfram-Schauerte, M

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