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GSH as intracellular copper chelator The most fundamental protective mechanism of GSH against cuproptosis is its function as an intracellular copper chelator

Appl Environ Microbiol 75(24):76317638 Hershko A, Ciechanover A (1998) The ubiquitin system

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Moreover, several clinical studies have identified elevated levels of numerous inflammatory markers, including CRP, TNF-, IL-1RA, sTNFR1, and sTNFR2, along with elevated serum NF-B levels, in patients with DPN compared to patients with T2DM without neuropathy, suggesting that patients with DPN exhibit a more pronounced inflammatory response (9698)