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Proc Natl Acad Sci U S A 98:1444014445 Lagirand-Cantaloube J, Offner N, Csibi A, Leibovitch MP, Batonnet-Pichon S, Tintignac LA, Segura CT, Leibovitch SA (2008) The initiation factor eIF3-f is a major target for atrogin1/MAFbx function in skeletal muscle atrophy

Carnitin-Stoffwechsel: Aufnahme von Carnitin: Das meiste in Lebensmitteln vorkommende Carnitin (54-86%) wird vom Dnndarm aufgenommen und tritt dort in den Blutkreislauf ein

Back to Archived Journals Research and Reports in Neonatology Volume 6 Screening for medium-chain acyl CoA dehydrogenase deficiency: current perspectives Authors Soler-Alfonso C, Bennett M, Ficicioglu C Received 3 June 2015 Accepted for publication 16 September 2015 Published 18 January 2016 Volume 2016:6 Pages 110 DOI Checked for plagiarism Yes Review by Single anonymous peer review Peer reviewer comments 5 Editor who approved publication: Dr Robert Schelonka Claudia Soler-Alfonso, 1 Michael J Bennett, 2 Can Ficicioglu 1 1 Department of Pediatrics, Section of Metabolic Disease, 2 Department of Pathology and Laboratory Medicine, The Childrens Hospital of Philadelphia and Perelman School of Medicine at the University of Pennsylvania, Philadelphia, PA, USA Abstract: Medium-chain acyl-CoA dehydrogenase (MCAD) deficiency is the most common disorder associated with fatty acid oxidation

Serum ALC was negatively correlated with LDL-C in hypercholesterolemic individuals Metabolomic qualitative and quantitative assessments of the patients plasma and clinical characteristics revealed that plasma ALC was significantly downregulated