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3ad), implying that CCl 4 -induced p53-dependent cell premature senescence were improved by IGF-1 gene therapy

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Conversely, FA disassembly is promoted by the loss of tension and controlled by a variety of pathways including microtubule-dependent 13,14 delivery of proteases for secretion 15 , endocytosis 13,16,17,18 and autophagy of FA components 19,20

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Turner syndrome in childhood and adolescence

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