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Pathological immobilisation of iron by protein aggregates such as amyloid and tau may sequester iron in a non-bioavailable form, or impaired autophagy may prevent efficient breakdown of ferritin to mobilise iron, thereby impairing mitochondrial ATP production and subsequently glutathione synthesis, an essential component of the ferroptosis defence system [29]

Endocr Dev (2013) 25:1624

Documentation Shortfalls Medical practices often overlook the need to clearly document how the evaluation and management (E/M) service is separate from the injection procedure during the same visit

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