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In fact, even if two people had the exact same tissue damage, their degree of pain could range from none to severe

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Our results show that depletion of macrophages from cardiac muscle strips not only prevented TLR2 activation-induced IL-1 release, but also the Pam3/TLR2/IL-1-induced prolongation of the cardiac AP, pointing to macrophages as the source of IL-1 secretion