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As shown in Figure 4, GHK-Cu significantly inhibited the LPS-induced phosphorylation of p38 MAPK and slightly decreased phosphorylation of JNK1/2 whereas the phosphorylation of ERK1/2 was not affected

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it can be administered via IV, IM, or SubQ injection

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Compared with FeSAN, the redox peak intensity of TMB in the presence of pFeSAN was much stronger, indicating more available active sites of pFeSAN for more efficient TMB oxidation

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While both are frequently referenced in similar research areas, they differ in their mechanisms and primary areas of investigation

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