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Description
As shown in Figure 4, GHK-Cu significantly inhibited the LPS-induced phosphorylation of p38 MAPK and slightly decreased phosphorylation of JNK1/2 whereas the phosphorylation of ERK1/2 was not affected
it can be administered via IV, IM, or SubQ injection

Compared with FeSAN, the redox peak intensity of TMB in the presence of pFeSAN was much stronger, indicating more available active sites of pFeSAN for more efficient TMB oxidation
While both are frequently referenced in similar research areas, they differ in their mechanisms and primary areas of investigation
