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Through these combined effects, ApoE4 promotes mitochondrial instability and defective mitochondrial turnover, ultimately exacerbating neuronal vulnerability and disease progression (Chen H

However, excessive ROS accumulation can lead to oxidative stress and damage to proteins, lipids and DNA, which results in cellular injury and potentially cell death

To further validate this phenomenon, we constructed a lens-induced myopia mouse model, which also displayed heightened Fe 2+ and lipid peroxidation levels in the highly myopic lens epithelia compared with the contralateral lens epithelia (Fig