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POS has been proposed as a probable mechanism contributing to systemic OxS, T2D progression, and vascular damage (see Figure 3) [91,92]
(2018) demonstrated that TET enzyme activity is directly dependent on vitamin C availability, establishing a mechanistic link between vitamin C status and epigenetic ageing that fundamentally repositions this nutrient beyond its antioxidant role
The role of survivin and Bcl-2 in zinc-induced apoptosis in prostate cancer cells
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